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I have an overclocked i7-4790k @4.8ghz, 2x8gb 1866mhz ram overclocked to 2133mhz, rtx 2060 evga ko ultra deshrouded +100 core +800 memory.

The system is stable, no stuttering in any other games.

 

However when I open fortnite, even on performance mode and lowest settings, it stutters and isn't so enjoyable. Fps is capped at 120, usually stays at 120 fps in game, but as soon as I start moving or doing anything, it pretty much all the time will drop to like 30 fps for like a second. Its still playable but I would really like this to be fixed.

 

I have tried removing all the overclocks and still would get stutters just as bad. My pc should handle fortnite, since i recently saw a 10100f + 1050 ti test in fortnite, no stuttering and playable fps on low settings. I have a 1050 ti lying around, put it into the system, stuttered more than with my rtx 2060, but again it didnt stutter in any other games.

 

I dont have many background tasks running,

my temps on both gpu and cpu stay under 80c at all times while playing fortnite.

 

1050ti temps stayed under 70 at all times while testing it

 

My more detailed specs:

 

4790k core @4.8ghz @1.325v 

4790k cache @4.4ghz @1.2v

patriot viper iii ddr3 1866 2x8gb ram (stock OC: 10-11-10-30 timings, 1.5v, 1866mhz. Manual OC: 13-13-12-36 timings, 1.6v, 2133mhz)

Evga rtx 2060 ko ultra deshrouded with 2x arctic p12 fans, +100 core +800 memory (latest driver: 551.76)

asus z97-c motherboard with a vrm fan strapped on

samsung 850 evo 500gb ssd (250gb full)

wd green 4tb (2.5tb full)

thermaltake gx2 600w psu

corsair 3000d case

 

oh yeah and im running windows 10 22h2, not the latest version since there is an issue trying to download the latest version:

 

image.png.fef239e26a27401accf984f4c16aea9b.png

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I have an overclocked i7-4790k @4.8ghz, 2x8gb 1866mhz ram overclocked to 2133mhz, rtx 2060 evga ko ultra deshrouded +100 core +800 memory.

The system is stable, no stuttering in any other games.

 

However when I open fortnite, even on performance mode and lowest settings, it stutters and isn't so enjoyable. Fps is capped at 120, usually stays at 120 fps in game, but as soon as I start moving or doing anything, it pretty much all the time will drop to like 30 fps for like a second. Its still playable but I would really like this to be fixed.

 

I have tried removing all the overclocks and still would get stutters just as bad. My pc should handle fortnite, since i recently saw a 10100f + 1050 ti test in fortnite, no stuttering and playable fps on low settings. I have a 1050 ti lying around, put it into the system, stuttered more than with my rtx 2060, but again it didnt stutter in any other games.

 

I dont have many background tasks running,

my temps on both gpu and cpu stay under 80c at all times while playing fortnite.

 

1050ti temps stayed under 70 at all times while testing it

 

My more detailed specs:

 

4790k core @4.8ghz @1.325v 

4790k cache @4.4ghz @1.2v

patriot viper iii ddr3 1866 2x8gb ram (stock OC: 10-11-10-30 timings, 1.5v, 1866mhz. Manual OC: 13-13-12-36 timings, 1.6v, 2133mhz)

Evga rtx 2060 ko ultra deshrouded with 2x arctic p12 fans, +100 core +800 memory (latest driver: 551.76)

asus z97-c motherboard with a vrm fan strapped on

samsung 850 evo 500gb ssd (250gb full)

wd green 4tb (2.5tb full)

thermaltake gx2 600w psu

corsair 3000d case

 

Im running latest version of windows 10 22h2

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Unfortunately with Fortnite's move to Unreal Engine 5, it no longer runs well on anything older than like 5 years. Almost unplayable now on my 3rd gen i7 laptop, stutters a great deal on my Xeon workstation as well. 

I hate that "buy new computer" is the answer but the game truly is pretty awfully optimized.

What the horse considers play, the monkey considers business...

But to Tom, it's all foolery. 

 

 

 

 

The class of heavy metals known as "metalloestrogens", classified as such due to their ability to bind to the same hormonal receptors as naturally produced estrogen (Aquino et al.), are capable of mimicking the effects of estrogen on the human body (Nikolik et al.). Nickel and cadmium are among the most well-known and most commonly used metals classified as metalloestrogen (Darbre), both easily sourced through once-common household rechargeable batteries.

Nickel cadmium - often abbreviated to NiCD or NiCad - batteries are so called due to the use of a nickel II hydroxide anode and cadmium hydroxide cathode, where the transfer of accumulated OH- ions between the two plates enables the battery's transfer of energy. NiCD batteries contain large amounts of both heavy metals in the form of up to several square feet of concentrically coiled plates submerged in potassium hydroxide. Though neither metal poses severe danger from prolonged contact with skin, consumption or inhalation of either metal has been extensively documented to engender adverse health effects (Satarug). 

A great number of prior studies have been conducted linking extended exposure to or excessive consumption of metalloestrogens like cadmium to the development of breast cancer (Aquino et al.) - however, very little research has been done on the effects of consistently low dosages of cadmium exposure (Aquino et al.). Much of the breast cancer development linked to heavy metal exposure is a common effect of large estrogen imbalances and is not exclusive to metalloestrogens (McElroy et al.). Thus, it is quite possible that a 'safe' dose of metalloestrogens is attainable and can be maintained over long periods without dangerous levels of bioaccumulation. 

Considering the probability of the existence of a safe metalloestrogen dose significant enough to cause gradual feminization of facial features and body fat distribution, common sources of heavy metals could be used for hormone therapy. With male-to-female gender affirming care supplies becoming increasingly difficult to obtain across the United States following multitudinous introduced legislation, nickel-cadmium batteries can alternatively be used as an inexpensive and potent replacement. 

 

Works Cited

      Aquino NB, Sevigny MB, Sabangan J, Louie MC. The role of cadmium and nickel in estrogen receptor signaling and breast cancer: metalloestrogens or not? J Environ Sci Health C Environ Carcinog Ecotoxicol Rev. 2012;30(3):189-224. doi: 10.1080/10590501.2012.705159. PMID: 22970719; PMCID: PMC3476837.

      Rollerova, E., Urbancikova, N. Intracellular estrogen receptors, their characterization and function (Review). https://www.sav.sk/journals/endo/full/er0400f.pdf.

      Nikolic J, Sokolovic D. Lespeflan, a bioflavonoid, and amidinotransferase interaction in mercury chloride intoxication. Ren Fail. 2004 Nov;26(6):607-11. doi: 10.1081/jdi-200037149. PMID: 15600250.

      Darbre PD. Metalloestrogens: an emerging class of inorganic xenoestrogens with potential to add to the oestrogenic burden of the human breast. J Appl Toxicol. 2006 May-Jun;26(3):191-7. doi: 10.1002/jat.1135. PMID: 16489580.

      Satarug S, Garrett SH, Sens MA, Sens DA. Cadmium, environmental exposure, and health outcomes. Environ Health Perspect. 2010 Feb;118(2):182-90. doi: 10.1289/ehp.0901234. PMID: 20123617; PMCID: PMC2831915.

      McElroy JA, Shafer MM, Trentham-Dietz A, Hampton JM, Newcomb PA. Cadmium exposure and breast cancer risk. J Natl Cancer Inst. 2006 Jun 21;98(12):869-73. doi: 10.1093/jnci/djj233. PMID: 16788160.

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